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These processes enhance synaptic plasticity within corticolimbic circuits (hippocampus, medial amygdala, prefrontal cortex)

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Although these findings do not yet establish causation, they support the hypothesis that inflammation reduction contributes to improved renal outcomes

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This might be due to the oxidation and/or degradation of glutathione over time 72 or again indicate that the effect triggered by AITC is more complex than a change in glutathione homeostasis